miR-148a is upregulated by Twist1 and T-bet and promotes Th1-cell survival by regulating the proapoptotic gene Bim

نویسندگان

  • Claudia Haftmann
  • Anna-Barbara Stittrich
  • Jakob Zimmermann
  • Zhuo Fang
  • Kristyna Hradilkova
  • Markus Bardua
  • Kerstin Westendorf
  • Gitta A Heinz
  • René Riedel
  • Julia Siede
  • Katrin Lehmann
  • Esther E Weinberger
  • David Zimmel
  • Uta Lauer
  • Thomas Häupl
  • Joachim Sieper
  • Marina Backhaus
  • Christian Neumann
  • Ute Hoffmann
  • Martina Porstner
  • Wei Chen
  • Joachim R Grün
  • Ria Baumgrass
  • Mareen Matz
  • Max Löhning
  • Alexander Scheffold
  • Jürgen Wittmann
  • Hyun-Dong Chang
  • Nikolaus Rajewsky
  • Hans-Martin Jäck
  • Andreas Radbruch
  • Mir-Farzin Mashreghi
چکیده

Repeatedly activated T helper 1 (Th1) cells present during chronic inflammation can efficiently adapt to the inflammatory milieu, for example, by expressing the transcription factor Twist1, which limits the immunopathology caused by Th1 cells. Here, we show that in repeatedly activated murine Th1 cells, Twist1 and T-bet induce expression of microRNA-148a (miR-148a). miR-148a regulates expression of the proapoptotic gene Bim, resulting in a decreased Bim/Bcl2 ratio. Inhibition of miR-148a by antagomirs in repeatedly activated Th1 cells increases the expression of Bim, leading to enhanced apoptosis. Knockdown of Bim expression by siRNA in miR-148a antagomir-treated cells restores viability of the Th1 cells, demonstrating that miR-148a controls survival by regulating Bim expression. Thus, Twist1 and T-bet not only control the differentiation and function of Th1 cells, but also their persistence in chronic inflammation.

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عنوان ژورنال:

دوره 45  شماره 

صفحات  -

تاریخ انتشار 2015